Promotes cell death. Successfully competes for the binding to Bcl-X(L), Bcl-2 and Bcl-W, thereby affecting the level of heterodimerization of these proteins with BAX. Can reverse the death repressor activity of Bcl-X(L), but not that of Bcl-2 (By similarity). Appears to act as a link between growth factor receptor signaling and the apoptotic pathways.

Western

Western blot analysis of Phospho-Bad (S112) in (1) HeLa lysates; (2) HeLa treated with Calcyculin A lysates using Phospho-Bad (Ser112) antibody.

-25 ~ -15℃保存，收到货之后有效期1年，避免反复冻融。